Adaptation to stress situations and physical loads
The book is devoted to the mechanism of adaptation of the organism to physical stress and stress situations, the use of this adaptation and its chemical “mediators” for the prevention and treatment of noncommunicable diseases, which represent an open challenge to modern medicine. Justified new view of stress-limiting systems of the organism and it is shown that
with the help of metabolites of these systems and their synthetic analogues can be successfully alerted diverse stress damage the body — from ulcerative lesions of the gastric mucosa to cardiac arrhythmias and ventricular fibrillation in myocardial infarction.
The mechanism of adaptation to physical loads
The main stages of adaptation to physical loads. Structural “footprint” of adaptation.
Protective effects of adaptation to physical loads. “Price” adaptation
Prevention of stress damage.
Prevention of ischemic heart damage.
Reduction of risk factors for cardiovascular disease.
Treatment and rehabilitation.
The similarity of positive cross-effects of adaptation to high-altitude hypoxia and physical exercise.
Negative cross-effects of adaptation.
Adaptation to stress situations and its protective effects
Damaging stress situation and adapting to it.
Pathogenesis of stress damage to the heart and preventing them through adaptation.
Stress the violation of antitumor immunity and its a warning by pre-adaptation.
The mechanism of adaptation to stress situations and stress – limiting systems of the organism
The main changes of neurohumoral regulation during adaptation to repeated stress.
Stress-limiting systems of the organism.
Cross-effects of adaptation to stress situations.
Prevention of fibrillation of the heart by means of adaptation to stress situations and other environmental factors
Stress in the etiology and pathogenesis of coronary heart disease.
Disorders of the nervous regulation in the pathogenesis of ventricular fibrillation and acute cardiac death.
Warning arrhythmias and fibrillation of the heart by means of adaptation to stress situations, physical stress and high-altitude hypoxia.
Prevention of cardiac arrhythmia by means of metabolites and activators of stress-limiting systems
Activators the GABAergic system and synthetic analogs of serotonin.
Free-radical oxidation in the pathogenesis of ischemic stress and myocardial injury and cardioprotective effect of antioxidants.
Free-radical oxidation in the pathogenesis of arrhythmia and preventing ventricular fibrillation in antioxidants.
The list of references
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